Hunger drive is controlled by hypothalamus. Glucostatic thery of hunger suggests that an increase in blood glucose level increases the activity of the safety center and decreases the activity of hunger center. Low blood glucose has the opposite effects. Aminoacid levels and lipid levels have also been suggested to influence hunger and satiety. The hepotostatic theory of hunger control argues for a role of liver as an important sensor and modulator of the body energy stores. digestion
glucostatic
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